Antibody data
- Antibody Data
- Antigen structure
- References [4]
- Comments [0]
- Validations
- Immunohistochemistry [4]
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- Product number
- NBP1-26549 - Provider product page
- Provider
- Novus Biologicals
- Proper citation
- Novus Cat#NBP1-26549, RRID:AB_1853202
- Product name
- Goat Polyclonal Collagen IV alpha 1 Antibody
- Antibody type
- Polyclonal
- Description
- Immunogen affinity purified. Reacts with conformational determinants on human type IV collagen as demonstrated by ELISA. May react with type IV collagen from other species. Exhibits
- Reactivity
- Human, Mouse, Rat, Bovine, Simian
- Host
- Goat
- Isotype
- IgG
- Vial size
- 0.1 mg
- Concentration
- 0.4 mg/ml
- Storage
- Store at 4C. Do not freeze.
Submitted references Optimal Mass Transport with Lagrangian Workflow Reveals Advective and Diffusion Driven Solute Transport in the Glymphatic System.
A type IV collagenase inhibitor, N-hydroxy-3-phenyl-2-(4-phenylbenzenesulfonamido) propanamide (BiPS), suppresses skin injury induced by sulfur mustard.
A Fully Automated High-Throughput Bioreactor System for Lung Regeneration.
Polidocanol induced tubal occlusion in nonhuman primates: immunohistochemical detection of collagen I-V.
Koundal S, Elkin R, Nadeem S, Xue Y, Constantinou S, Sanggaard S, Liu X, Monte B, Xu F, Van Nostrand W, Nedergaard M, Lee H, Wardlaw J, Benveniste H, Tannenbaum A
Scientific reports 2020 Feb 6;10(1):1990
Scientific reports 2020 Feb 6;10(1):1990
A type IV collagenase inhibitor, N-hydroxy-3-phenyl-2-(4-phenylbenzenesulfonamido) propanamide (BiPS), suppresses skin injury induced by sulfur mustard.
Chang YC, Hahn RA, Gordon MK, Laskin JD, Gerecke DR
Toxicology and applied pharmacology 2020 Aug 15;401:115078
Toxicology and applied pharmacology 2020 Aug 15;401:115078
A Fully Automated High-Throughput Bioreactor System for Lung Regeneration.
Gorman DE, Wu T, Gilpin SE, Ott HC
Tissue engineering. Part C, Methods 2018 Nov;24(11):671-678
Tissue engineering. Part C, Methods 2018 Nov;24(11):671-678
Polidocanol induced tubal occlusion in nonhuman primates: immunohistochemical detection of collagen I-V.
Slayden OD, Lee DO, Yao S, Jensen JT
Contraception 2016 Nov;94(5):521-526
Contraception 2016 Nov;94(5):521-526
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Supportive validation
- Submitted by
- Novus Biologicals (provider)
- Main image
- Experimental details
- Immunohistochemistry-Paraffin: Collagen IV alpha 1 Antibody [NBP1-26549] - Human gastric cancer tissue was stained with Goat IgG-UNLB isotype control (left) and Goat Anti-Type IV Collagen-UNLB; (right) followed by Swine Anti-Goat IgG(H+L), Human/Rat/Mouse SP ads-HRP DAB, and hematoxylin.
- Submitted by
- Novus Biologicals (provider)
- Main image
- Experimental details
- Immunohistochemistry-Frozen: Collagen IV alpha 1 Antibody [NBP1-26549] - Frozen sections of E12.5 eyes from Col4a1+/+ mice were stained with Goat Anti-Type IV Collagen-UNLB followed by a secondary antibody and DAPI. Image from Mao M, Kiss M, Ou Y, Gould DB. Genetic dissection of anterior segment dysgenesis caused by a Col4a1 mutation. Dis Model Mech. 2017 Feb 24. doi: 10.1242/dmm.027888.
- Submitted by
- Novus Biologicals (provider)
- Main image
- Experimental details
- Immunohistochemistry-Frozen: Collagen IV alpha 1 Antibody [NBP1-26549] - Frozen Itga3-/-/Gpr56-/- E11.5 mouse neocortex section was stained with Goat Anti-Type IV Collagen-UNLB anti-Tuj1, and anti-Nestin followed by secondary antibodies. Image from Jeong S, Luo R, Singer K, Giera S, Kreidberg J, Kiyozumi D, et al. GPR56 functions together with alpha 3beta1 integrin in regulating cerebral cortical development. PLoS One. 2013;8(7):e68781.
- Submitted by
- Novus Biologicals (provider)
- Main image
- Experimental details
- Immunohistochemistry-Frozen: Collagen IV alpha 1 Antibody [NBP1-26549] - Frozen rat kidney section following SNX was stained with Goat Anti-Type IV Collagen-UNLB followed by a secondary antibody and DAB. Image from Yuen DA, Connelly KA, Advani A, Liao C, Kuliszewski MA, Trogadis J, et al. Culture-modified bone marrow cells attenuate cardiac and renal injury in a chronic kidney disease rat model via a novel antifibrotic mechanism. PLoS One. 2010;5(3):e9543.