Antibody data
- Antibody Data
- Antigen structure
- References [4]
- Comments [0]
- Validations
- Western blot [1]
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Validation data
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- Product number
- ABIN1450013 - Provider product page
- Provider
- antibodies-online
- Product name
- anti-Amyloid beta (A4) Precursor Protein-Binding, Family A, Member 1 (APBA1) (N-Term) antibody
- Antibody type
- Polyclonal
- Antigen
- Other
- Reactivity
- Human, Mouse, Rat
- Host
- Rabbit
- Isotype
- IgG
- Vial size
- 0.1 mg
Submitted references The molecular basis of the Caskin1 and Mint1 interaction with CASK.
Modulation of amyloid precursor protein metabolism by X11alpha /Mint-1. A deletion analysis of protein-protein interaction domains.
Association of neuronal calcium channels with modular adaptor proteins.
The phosphotyrosine interaction domains of X11 and FE65 bind to distinct sites on the YENPTY motif of amyloid precursor protein.
Stafford RL, Ear J, Knight MJ, Bowie JU
Journal of molecular biology 2011 Sep 9;412(1):3-13
Journal of molecular biology 2011 Sep 9;412(1):3-13
Modulation of amyloid precursor protein metabolism by X11alpha /Mint-1. A deletion analysis of protein-protein interaction domains.
Mueller HT, Borg JP, Margolis B, Turner RS
The Journal of biological chemistry 2000 Dec 15;275(50):39302-6
The Journal of biological chemistry 2000 Dec 15;275(50):39302-6
Association of neuronal calcium channels with modular adaptor proteins.
Maximov A, Südhof TC, Bezprozvanny I
The Journal of biological chemistry 1999 Aug 27;274(35):24453-6
The Journal of biological chemistry 1999 Aug 27;274(35):24453-6
The phosphotyrosine interaction domains of X11 and FE65 bind to distinct sites on the YENPTY motif of amyloid precursor protein.
Borg JP, Ooi J, Levy E, Margolis B
Molecular and cellular biology 1996 Nov;16(11):6229-41
Molecular and cellular biology 1996 Nov;16(11):6229-41
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Supportive validation
- Submitted by
- antibodies-online (provider)
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- Experimental details
- Western blot analysis of APBA1 in rat brain tissue lysate with APBA1 antibody Cat.-No AP55509PU-N at 0.5 μg/ml in (A) the absence and (B) the presence of blocking peptide.