Antibody data
- Antibody Data
- Antigen structure
- References [7]
- Comments [0]
- Validations
- Western blot [1]
- Immunohistochemistry [1]
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Validation data
Reference
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- Product number
- 13122-1-AP - Provider product page
- Provider
- Proteintech Group
- Proper citation
- Proteintech Cat#13122-1-AP, RRID:AB_10638904
- Product name
- ACADSB antibody
- Antibody type
- Polyclonal
- Description
- KD/KO validated ACADSB antibody (Cat. #13122-1-AP) is a rabbit polyclonal antibody that shows reactivity with human, mouse, rat and has been validated for the following applications: IHC, WB, ELISA.
- Reactivity
- Human, Mouse, Rat
- Host
- Rabbit
- Conjugate
- Unconjugated
- Isotype
- IgG
- Vial size
- 20ul, 150ul
Submitted references Icariside II prevents kidney fibrosis development in chronic kidney disease by promoting fatty acid oxidation.
Comparison of the stage-dependent mitochondrial changes in response to pressure overload between the diseased right and left ventricle in the rat.
AMPK induces degradation of the transcriptional repressor PROX1 impairing branched amino acid metabolism and tumourigenesis.
Mitochondrial FAD shortage in SLC25A32 deficiency affects folate-mediated one-carbon metabolism.
Global mitochondrial protein import proteomics reveal distinct regulation by translation and translocation machinery.
Decreased Expression of ACADSB Predicts Poor Prognosis in Clear Cell Renal Cell Carcinoma.
ACADSB regulates ferroptosis and affects the migration, invasion, and proliferation of colorectal cancer cells.
Wang M, Wang J, Wang L, Feng X, Qian Y, Ye C, Wang C
Phytotherapy research : PTR 2024 Feb;38(2):839-855
Phytotherapy research : PTR 2024 Feb;38(2):839-855
Comparison of the stage-dependent mitochondrial changes in response to pressure overload between the diseased right and left ventricle in the rat.
Li L, Niemann B, Knapp F, Werner S, Mühlfeld C, Schneider JP, Jurida LM, Molenda N, Schmitz ML, Yin X, Mayr M, Schulz R, Kracht M, Rohrbach S
Basic research in cardiology 2024 Aug;119(4):587-611
Basic research in cardiology 2024 Aug;119(4):587-611
AMPK induces degradation of the transcriptional repressor PROX1 impairing branched amino acid metabolism and tumourigenesis.
Wang Y, Luo M, Wang F, Tong Y, Li L, Shu Y, Qiao K, Zhang L, Yan G, Liu J, Ji H, Xie Y, Zhang Y, Gao WQ, Liu Y
Nature communications 2022 Nov 24;13(1):7215
Nature communications 2022 Nov 24;13(1):7215
Mitochondrial FAD shortage in SLC25A32 deficiency affects folate-mediated one-carbon metabolism.
Peng MZ, Shao YX, Li XZ, Zhang KD, Cai YN, Lin YT, Jiang MY, Liu ZC, Su XY, Zhang W, Jiang XL, Liu L
Cellular and molecular life sciences : CMLS 2022 Jun 21;79(7):375
Cellular and molecular life sciences : CMLS 2022 Jun 21;79(7):375
Global mitochondrial protein import proteomics reveal distinct regulation by translation and translocation machinery.
Schäfer JA, Bozkurt S, Michaelis JB, Klann K, Münch C
Molecular cell 2022 Jan 20;82(2):435-446.e7
Molecular cell 2022 Jan 20;82(2):435-446.e7
Decreased Expression of ACADSB Predicts Poor Prognosis in Clear Cell Renal Cell Carcinoma.
Liu X, Zhang W, Wang H, Zhu L, Xu K
Frontiers in oncology 2021;11:762629
Frontiers in oncology 2021;11:762629
ACADSB regulates ferroptosis and affects the migration, invasion, and proliferation of colorectal cancer cells.
Lu D, Yang Z, Xia Q, Gao S, Sun S, Luo X, Li Z, Zhang X, Li X
Cell biology international 2020 Nov;44(11):2334-2343
Cell biology international 2020 Nov;44(11):2334-2343
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Supportive validation
- Submitted by
- Proteintech Group (provider)
- Main image
- Experimental details
- HEK-293 cells were subjected to SDS PAGE followed by western blot with 13122-1-AP(ACADSB antibody) at dilution of 1:300
- Sample type
- cell line
Supportive validation
- Submitted by
- Proteintech Group (provider)
- Main image
- Experimental details
- The ACADSB antibody from Proteintech is a rabbit polyclonal antibody to a recombinant protein of human ACADSB. This antibody recognizes human, mouse, rat antigen. The ACADSB antibody has been validated for the following applications: ELISA, WB, IHC analysis.