Antibody data
- Antibody Data
- Antigen structure
- References [4]
- Comments [0]
- Validations
- Western blot [1]
- Immunohistochemistry [1]
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- Product number
- ABIN2842053 - Provider product page
- Provider
- antibodies-online
- Product name
- anti-Nuclear Receptor Subfamily 2, Group C, Member 2 (NR2C2) (AA 574-606), (C-Term) antibody
- Antibody type
- Polyclonal
- Description
- This antibody is prepared by Saturated Ammonium Sulfate (SAS) precipitation followed by dialysis against PBS.
- Reactivity
- Human
- Host
- Rabbit
- Epitope
- AA 574-606, C-Term
- Antibody clone number
- RB0993
- Vial size
- 80 μL
- Storage
- Maintain refrigerated at 2-8°C for up to 6 months. For long term storage store at -20°C in small aliquots to prevent freeze-thaw cycles.
Submitted references Tumor necrosis factor-alpha-induced IKK phosphorylation of NF-kappaB p65 on serine 536 is mediated through the TRAF2, TRAF5, and TAK1 signaling pathway.
Feedback control of the protein kinase TAK1 by SAPK2a/p38alpha.
A dominant negative TAK1 inhibits cellular fibrotic responses induced by TGF-beta.
Transforming growth factor-beta1 (TGF-beta)-induced apoptosis of prostate cancer cells involves Smad7-dependent activation of p38 by TGF-beta-activated kinase 1 and mitogen-activated protein kinase kinase 3.
Sakurai H, Suzuki S, Kawasaki N, Nakano H, Okazaki T, Chino A, Doi T, Saiki I
The Journal of biological chemistry 2003 Sep 19;278(38):36916-23
The Journal of biological chemistry 2003 Sep 19;278(38):36916-23
Feedback control of the protein kinase TAK1 by SAPK2a/p38alpha.
Cheung PC, Campbell DG, Nebreda AR, Cohen P
The EMBO journal 2003 Nov 3;22(21):5793-805
The EMBO journal 2003 Nov 3;22(21):5793-805
A dominant negative TAK1 inhibits cellular fibrotic responses induced by TGF-beta.
Ono K, Ohtomo T, Ninomiya-Tsuji J, Tsuchiya M
Biochemical and biophysical research communications 2003 Jul 25;307(2):332-7
Biochemical and biophysical research communications 2003 Jul 25;307(2):332-7
Transforming growth factor-beta1 (TGF-beta)-induced apoptosis of prostate cancer cells involves Smad7-dependent activation of p38 by TGF-beta-activated kinase 1 and mitogen-activated protein kinase kinase 3.
Edlund S, Bu S, Schuster N, Aspenström P, Heuchel R, Heldin NE, ten Dijke P, Heldin CH, Landström M
Molecular biology of the cell 2003 Feb;14(2):529-44
Molecular biology of the cell 2003 Feb;14(2):529-44
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Supportive validation
- Submitted by
- antibodies-online (provider)
- Main image
- Experimental details
- WB
Supportive validation
- Submitted by
- antibodies-online (provider)
- Main image
- Experimental details
- IHC