Antibody data
- Antibody Data
- Antigen structure
- References [8]
- Comments [0]
- Validations [0]
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- Product number
- 12161-1-AP - Provider product page
- Provider
- Proteintech Group
- Product name
- PTPRO antibody
- Antibody type
- Polyclonal
- Description
- PTPRO antibody (Cat. #12161-1-AP) is a rabbit polyclonal antibody that shows reactivity with human, mouse, rat and has been validated for the following applications: ELISA.
- Reactivity
- Human, Mouse, Rat
- Host
- Rabbit
- Conjugate
- Unconjugated
- Isotype
- IgG
- Vial size
- 20ul, 150ul
Submitted references MiRNA-548c-5p downregulates inflammatory response in preeclampsia via targeting PTPRO.
PTPRO Promotes Oxidized Low-Density Lipoprotein Induced Oxidative Stress and Cell Apoptosis through Toll-Like Receptor 4/Nuclear Factor κB Pathway.
Aggravated Liver Injury but Attenuated Inflammation in PTPRO-Deficient Mice Following LPS/D-GaIN Induced Fulminant Hepatitis.
PTPRO-associated hepatic stellate cell activation plays a critical role in liver fibrosis.
PTPRO-mediated autophagy prevents hepatosteatosis and tumorigenesis.
Interaction of PTPRO and TLR4 signaling in hepatocellular carcinoma.
PTPRO plays a dual role in hepatic ischemia reperfusion injury through feedback activation of NF-κB.
Estrogen-sensitive PTPRO expression represses hepatocellular carcinoma progression by control of STAT3.
Wang Z, Wang P, Wang Z, Qin Z, Xiu X, Xu D, Zhang X, Wang Y
Journal of cellular physiology 2019 Jul;234(7):11149-11155
Journal of cellular physiology 2019 Jul;234(7):11149-11155
PTPRO Promotes Oxidized Low-Density Lipoprotein Induced Oxidative Stress and Cell Apoptosis through Toll-Like Receptor 4/Nuclear Factor κB Pathway.
Liang C, Wang X, Hu J, Lian X, Zhu T, Zhang H, Gu N, Li J
Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology 2017;42(2):495-505
Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology 2017;42(2):495-505
Aggravated Liver Injury but Attenuated Inflammation in PTPRO-Deficient Mice Following LPS/D-GaIN Induced Fulminant Hepatitis.
Wang X, Yan S, Xu D, Li J, Xie Y, Hou J, Jiang R, Zhang C, Sun B
Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology 2015;37(1):214-24
Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology 2015;37(1):214-24
PTPRO-associated hepatic stellate cell activation plays a critical role in liver fibrosis.
Zhang X, Tan Z, Wang Y, Tang J, Jiang R, Hou J, Zhuo H, Wang X, Ji J, Qin X, Sun B
Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology 2015;35(3):885-98
Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology 2015;35(3):885-98
PTPRO-mediated autophagy prevents hepatosteatosis and tumorigenesis.
Zhang W, Hou J, Wang X, Jiang R, Yin Y, Ji J, Deng L, Huang X, Wang K, Sun B
Oncotarget 2015 Apr 20;6(11):9420-33
Oncotarget 2015 Apr 20;6(11):9420-33
Interaction of PTPRO and TLR4 signaling in hepatocellular carcinoma.
Xu D, Wang X, Yan S, Yin Y, Hou J, Wang X, Sun B
Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine 2014 Oct;35(10):10267-73
Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine 2014 Oct;35(10):10267-73
PTPRO plays a dual role in hepatic ischemia reperfusion injury through feedback activation of NF-κB.
Hou J, Xia Y, Jiang R, Chen D, Xu J, Deng L, Huang X, Wang X, Sun B
Journal of hepatology 2014 Feb;60(2):306-12
Journal of hepatology 2014 Feb;60(2):306-12
Estrogen-sensitive PTPRO expression represses hepatocellular carcinoma progression by control of STAT3.
Hou J, Xu J, Jiang R, Wang Y, Chen C, Deng L, Huang X, Wang X, Sun B
Hepatology (Baltimore, Md.) 2013 Feb;57(2):678-88
Hepatology (Baltimore, Md.) 2013 Feb;57(2):678-88
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