Explore
Validate
Learn
Western blotAntibody data
- Antibody Data
- Antigen structure
- References [5]
- Comments [0]
- Validations
- Western blot [2]
- ELISA [1]
- Immunohistochemistry [1]
Submit
Validation data
Reference
Comment
Report error
- Product number
- ABIN393313 - Provider product page
- Provider
- antibodies-online
- Product name
- anti-Tripeptidyl Peptidase I (TPP1) (AA 195-305) antibody
- Antibody type
- Monoclonal
- Description
- This antibody is purified through a protein G column, eluted with high and low pH buffers and neutralized immediately, followed by dialysis against PBS.
- Reactivity
- Human
- Host
- Mouse
- Epitope
- AA 195-305
- Isotype
- IgG
- Antibody clone number
- 3B1
- Vial size
- 100 μg
- Storage
- Maintain refrigerated at 2-8°C for up to 6 months. For long term storage store at -20°C in small aliquots to prevent freeze-thaw cycles
Submitted references POT1-TPP1 enhances telomerase processivity by slowing primer dissociation and aiding translocation.
Gene therapy for late infantile neuronal ceroid lipofuscinosis: neurosurgical considerations.
Late infantile neuronal ceroid lipofuscinosis: a new mutation in Arabs.
Mutations in CLN7/MFSD8 are a common cause of variant late-infantile neuronal ceroid lipofuscinosis.
Lysosomal serine protease CLN2 regulates tumor necrosis factor-alpha-mediated apoptosis in a Bid-dependent manner.
Latrick CM, Cech TR
The EMBO journal 2010 Mar 3;29(5):924-33
The EMBO journal 2010 Mar 3;29(5):924-33
Gene therapy for late infantile neuronal ceroid lipofuscinosis: neurosurgical considerations.
Souweidane MM, Fraser JF, Arkin LM, Sondhi D, Hackett NR, Kaminsky SM, Heier L, Kosofsky BE, Worgall S, Crystal RG, Kaplitt MG
Journal of neurosurgery. Pediatrics 2010 Aug;6(2):115-22
Journal of neurosurgery. Pediatrics 2010 Aug;6(2):115-22
Late infantile neuronal ceroid lipofuscinosis: a new mutation in Arabs.
Goldberg-Stern H, Halevi A, Marom D, Straussberg R, Mimouni-Bloch A
Pediatric neurology 2009 Oct;41(4):297-300
Pediatric neurology 2009 Oct;41(4):297-300
Mutations in CLN7/MFSD8 are a common cause of variant late-infantile neuronal ceroid lipofuscinosis.
Kousi M, Siintola E, Dvorakova L, Vlaskova H, Turnbull J, Topcu M, Yuksel D, Gokben S, Minassian BA, Elleder M, Mole SE, Lehesjoki AE
Brain : a journal of neurology 2009 Mar;132(Pt 3):810-9
Brain : a journal of neurology 2009 Mar;132(Pt 3):810-9
Lysosomal serine protease CLN2 regulates tumor necrosis factor-alpha-mediated apoptosis in a Bid-dependent manner.
Autefage H, Albinet V, Garcia V, Berges H, Nicolau ML, Therville N, Altié MF, Caillaud C, Levade T, Andrieu-Abadie N
The Journal of biological chemistry 2009 Apr 24;284(17):11507-16
The Journal of biological chemistry 2009 Apr 24;284(17):11507-16
No comments: Submit comment
Supportive validation
- Submitted by
- antibodies-online (provider)
- Main image
- Experimental details
- WB
- Submitted by
- antibodies-online (provider)
- Main image
- Experimental details
- WB
Supportive validation
- Submitted by
- antibodies-online (provider)
- Main image
- Experimental details
- ELISA
Supportive validation
- Submitted by
- antibodies-online (provider)
- Main image
- Experimental details
- IHC
